Qiong-Hua Chen

No ORCID on file · 15 papers in corpus · active 2004-2025

Study types

  • article 8
  • other 5
  • dataset 2

Condition tags

  • endometriosis 14
  • mesh:D004715 10
  • infertility 2
  • dysmenorrhea 1
other 2025
Molecular medicine (Cambridge, Mass.) ·doi:10.1186/s10020-025-01130-8

BACKGROUND: Endometriosis (EMs) is a condition characterized by the growth of endometrial tissue outside the uterine cavity. Although this condition is benign, it has cancer-like features. N6-methyladenosine (m6A) is a common RNA modificati…

article 2020
Journal of cellular and molecular medicine ·doi:10.1111/jcmm.15689

Abstract Endometriosis is a benign gynaecological disease appearing with pelvic pain, rising dysmenorrhoea and infertility seriously impacting on 10% of reproductive‐age females. This research attempts to demonstrate the function and molecu…

article 2018
International journal of clinical and experimental pathology

Endometriosis is an estrogen-dependent disease associated with pain and infertility. The objective of this study was to determine the expression of ZEB1 in endometriosis and its role in 17β-estradiol (E2)-induced epithelial-mesenchymal tran…

other 2017
Journal of Obstetrics and Gynaecology Research ·doi:10.1111/jog.13201

AIM: Lipoxin A4 (LXA4 ) can function as an endogenous 'breaking signal' in inflammation and plays an important role in the progression of endometriosis. The proteome responses to interleukin-1β (IL-1β) or LXA4 in human endometriotic stromal…

article 2017
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719117718271
other 2017
·doi:10.25384/sage.c.4106783

Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…

other 2017
·doi:10.25384/sage.c.4106783.v1

Objective:Epithelial–mesenchymal transition (EMT) is essential for embryogenesis, fibrosis, and tumor metastasis. Aberrant EMT phenomenon has been reported in endometriotic tissues of patients with endometriosis (EM). In this study, we furt…

dataset 2017
·doi:10.25384/sage.6294656

Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…

dataset 2017
·doi:10.25384/sage.6294656.v1

Supplementary Material for Lipoxin A4 Suppresses Estrogen-Induced Epithelial–Mesenchymal Transition via ALXR-Dependent Manner in Endometriosis by Rong-Feng Wu, Zhi-Xiong Huang, Jing Ran, Song-Juan Dai, Dian-Chao Lin, Tai-Wei Ng, Qing-Xi Che…

article 2014
Fertility and sterility ·doi:10.1016/j.fertnstert.2014.03.029
other 2010
Fertility and sterility ·doi:10.1016/j.fertnstert.2009.02.013

OBJECTIVE: To examine the change in proinflammatory cytokines in the pathologic processes of endometriosis in mice. DESIGN: A dynamic study on a murine model of endometriosis. SETTING: Medical school. ANIMAL(S): Female BALB/c mice. INTE…

article 2010
Human Reproduction ·doi:10.1093/humrep/deq287

BACKGROUND: p38 mitogen-activated protein kinase (p38 MAPK), a regulator of inflammation, may play a role in the pathogenesis of endometriosis (EM). We studied the effect of SB203580, a p38 MAPK inhibitor, on the development of EM in a mous…

article 2010
Yao xue xue bao = Acta pharmaceutica Sinica

Endometriosis, an oestrogen-dependent disorder, is related to inflammation, p38 Mitogen activated protein kinases (p38 MAPK) can be activated by sex hormone and inflammatory factors, which plays an important role in many cellular reactions …

article 2009
Fertility and sterility ·doi:10.1016/j.fertnstert.2009.01.107
article 2004
Zhonghua fu chan ke za zhi

OBJECTIVE: To study the expression of matrix metalloproteinase-9(MMP-9) and tissue inhibitor of metalloproteinase-1 (TIMP-1) in ectopic and eutopic endometrium in patients with endometriosis. METHODS: The expressions of MMP-9 and TIMP-1 in …