Effect of active vitamin D on proliferation, cell cycle and apoptosis in endometriotic stromal cells

Nesa Rashidi; Soheila Arefi; Maryam Sadri; Ali-Akbar Delbandi; Ali‐Akbar Delbandi

doi:10.1016/j.rbmo.2022.11.009

Effect of active vitamin D on proliferation, cell cycle and apoptosis in endometriotic stromal cells

Nesa Rashidi, Soheila Arefi, Maryam Sadri, Ali-Akbar Delbandi, Ali‐Akbar Delbandi

Reproductive biomedicine online · 2022 · vol. 46(3) , pp. 436–445 · doi:10.1016/j.rbmo.2022.11.009 · PMID:36588053 · W4309293926

article OA: closed CC0 ⤵ 7 in-corpus citations

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⚙ AI-generated summary by claude@2026-06, 2026-06-08 ⓘ

This study investigated the impact of active vitamin D on the proliferation, cell cycle, and apoptosis of human endometriotic stromal cells.

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Condition tags

mesh:D004715

MeSH descriptors

Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Endometriosis Vitamin D Vitamin D Vitamin D Vitamin D Vitamin D Vitamin D Vitamin D

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References (68)

1,25‐Dihydroxy Vitamin D3 Modulates Endometriosis‐Related Features of Human Endometriotic Stromal Cells via openalex
1,25-Dihydroxyvitamin D3 Regulates Expression of Sex Steroid Receptors in Human Uterine Fibroid Cells via openalex
17βE2 promotes cell proliferation in endometriosis by decreasing PTEN via NFκB-dependent pathway via openalex
Abnormal activation of Ras/Raf/MAPK and RhoA/ROCKII signalling pathways in eutopic endometrial stromal cells of patients with endometriosis via openalex
Advances in the management of endometriosis: an update for clinicians via openalex
Apoptosis and bcl-2 expression in normal human endometrium, endometriosis and adenomyosis via openalex
Apoptosis and endometriosis via openalex
Apoptosis and expression of Bcl-2 and Bax in eutopic endometrium from women with endometriosis via openalex
Apoptosis in Endometriosis via openalex
Apoptosis in human endometrium and endometriosis via openalex
Current and emerging treatment options for endometriosis via openalex
Downregulation of CD36 results in reduced phagocytic ability of peritoneal macrophages of women with endometriosis via openalex
Effects of 1,25-Dihydroxy Vitamin D3 on Endometriosis via openalex
Endometrial Anomalies in Women with Endometriosis via openalex
Endometrial cells from women with endometriosis have increased adhesion and proliferative capacity in response to extracellular matrix components: towards a mechanistic model for endometriosis progression via openalex
Endometriosis: pathogenesis and treatment via openalex
Endometriotic cells are resistant to interferon-γ-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis via openalex
Estrogen signaling in the proliferative endometrium: implications in endometriosis via openalex
Eutopic and ectopic stromal cells from patients with endometriosis exhibit differential invasive, adhesive, and proliferative behavior via openalex
Growth mechanisms of endometriotic cells in implanted places: a review via openalex
Increased Mitogen-Activated Protein Kinase Kinase/Extracellularly Regulated Kinase Activity in Human Endometrial Stromal Fibroblasts of Women with Endometriosis Reduces 3′,5′-Cyclic Adenosine 5′-Monophosphate Inhibition of Cyclin D1 via openalex
miR-503, a microRNA epigenetically repressed in endometriosis, induces apoptosis and cell-cycle arrest and inhibits cell proliferation, angiogenesis, and contractility of human ovarian endometriotic stromal cells via openalex
Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity via openalex
Platelet-derived growth factor (PDGF), epidermal growth factor (EGF), and EGF and PDGF beta-receptors in human endometrial tissue: localization and in vitro action via openalex
Resveratrol reduces the expression of insulin‐like growth factor‐1 and hepatocyte growth factor in stromal cells of women with endometriosis compared with nonendometriotic women via openalex
Revised American Society for Reproductive Medicine classification of endometriosis: 1996 via openalex
Steroid regulation of menstrual bleeding and endometrial repair via openalex
The expression of estrogen receptors as well as GREB1, c-MYC, and cyclin D1, estrogen-regulated genes implicated in proliferation, is increased in peritoneal endometriosis via openalex
The Expression Status of G Protein-Coupled Receptor GPR30 Is Associated with the Clinical Characteristics of Endometriosis via openalex
Vitamin D and Endometrium: A Systematic Review of a Neglected Area of Research via openalex
Vitamin D deficiency as a risk factor for endometriosis in Iranian women via openalex
W3134885188 via openalex
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W6606722718 via openalex
W6665575137 via openalex
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W2165968193 via openalex
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W1993184229 via openalex
W1999861997 via openalex
W2006439158 via openalex
W2036524303 via openalex
W2038177382 via openalex
W2066888185 via openalex
W2075576602 via openalex
W2081646177 via openalex
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W2084375799 via openalex
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W2097444886 via openalex
W2100688315 via openalex
W2104036704 via openalex
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W2120343162 via openalex
W2121522838 via openalex
W2122908584 via openalex
W2132577372 via openalex
W2157568141 via openalex
W166362025 via openalex
W2322085293 via openalex
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W2891820668 via openalex
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W2980976550 via openalex
W3028984870 via openalex
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Cited by (7)

Source provenance

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License: CC0 · commercial use OK

[1] 1,25‐Dihydroxy Vitamin D3 Modulates Endometriosis‐Related Features of Human Endometriotic Stromal Cells via openalex

[2] 1,25-Dihydroxyvitamin D3 Regulates Expression of Sex Steroid Receptors in Human Uterine Fibroid Cells via openalex

[3] 17βE2 promotes cell proliferation in endometriosis by decreasing PTEN via NFκB-dependent pathway via openalex

[4] Abnormal activation of Ras/Raf/MAPK and RhoA/ROCKII signalling pathways in eutopic endometrial stromal cells of patients with endometriosis via openalex

[5] Advances in the management of endometriosis: an update for clinicians via openalex

[6] Apoptosis and bcl-2 expression in normal human endometrium, endometriosis and adenomyosis via openalex

[7] Apoptosis and endometriosis via openalex

[8] Apoptosis and expression of Bcl-2 and Bax in eutopic endometrium from women with endometriosis via openalex

[9] Apoptosis in Endometriosis via openalex

[10] Apoptosis in human endometrium and endometriosis via openalex

[11] Current and emerging treatment options for endometriosis via openalex

[12] Downregulation of CD36 results in reduced phagocytic ability of peritoneal macrophages of women with endometriosis via openalex

[13] Effects of 1,25-Dihydroxy Vitamin D3 on Endometriosis via openalex

[14] Endometrial Anomalies in Women with Endometriosis via openalex

[15] Endometrial cells from women with endometriosis have increased adhesion and proliferative capacity in response to extracellular matrix components: towards a mechanistic model for endometriosis progression via openalex

[16] Endometriosis: pathogenesis and treatment via openalex

[17] Endometriotic cells are resistant to interferon-γ-induced cell growth inhibition and apoptosis: a possible mechanism involved in the pathogenesis of endometriosis via openalex

[18] Estrogen signaling in the proliferative endometrium: implications in endometriosis via openalex

[19] Eutopic and ectopic stromal cells from patients with endometriosis exhibit differential invasive, adhesive, and proliferative behavior via openalex

[20] Growth mechanisms of endometriotic cells in implanted places: a review via openalex

[21] Increased Mitogen-Activated Protein Kinase Kinase/Extracellularly Regulated Kinase Activity in Human Endometrial Stromal Fibroblasts of Women with Endometriosis Reduces 3′,5′-Cyclic Adenosine 5′-Monophosphate Inhibition of Cyclin D1 via openalex

[22] miR-503, a microRNA epigenetically repressed in endometriosis, induces apoptosis and cell-cycle arrest and inhibits cell proliferation, angiogenesis, and contractility of human ovarian endometriotic stromal cells via openalex

[23] Peritoneal endometriosis due to the menstrual dissemination of endometrial tissue into the peritoneal cavity via openalex

[24] Platelet-derived growth factor (PDGF), epidermal growth factor (EGF), and EGF and PDGF beta-receptors in human endometrial tissue: localization and in vitro action via openalex

[25] Resveratrol reduces the expression of insulin‐like growth factor‐1 and hepatocyte growth factor in stromal cells of women with endometriosis compared with nonendometriotic women via openalex

[26] Revised American Society for Reproductive Medicine classification of endometriosis: 1996 via openalex

[27] Steroid regulation of menstrual bleeding and endometrial repair via openalex

[28] The expression of estrogen receptors as well as GREB1, c-MYC, and cyclin D1, estrogen-regulated genes implicated in proliferation, is increased in peritoneal endometriosis via openalex

[29] The Expression Status of G Protein-Coupled Receptor GPR30 Is Associated with the Clinical Characteristics of Endometriosis via openalex

[30] Vitamin D and Endometrium: A Systematic Review of a Neglected Area of Research via openalex

[31] Vitamin D deficiency as a risk factor for endometriosis in Iranian women via openalex

[32] W3134885188 via openalex

[33] W3197522732 via openalex

[34] W4211081176 via openalex

[35] W6606722718 via openalex

[36] W6665575137 via openalex

[37] W6769818433 via openalex

[38] W2165968193 via openalex

[39] W1969768367 via openalex

[40] W1980817286 via openalex

[41] W1993184229 via openalex

[42] W1999861997 via openalex

[43] W2006439158 via openalex

[44] W2036524303 via openalex

[45] W2038177382 via openalex

[46] W2066888185 via openalex

[47] W2075576602 via openalex

[48] W2081646177 via openalex

[49] W2084286096 via openalex

[50] W2084375799 via openalex

[51] W2088937747 via openalex

[52] W2097444886 via openalex

[53] W2100688315 via openalex

[54] W2104036704 via openalex

[55] W2108352304 via openalex

[56] W2120343162 via openalex

[57] W2121522838 via openalex

[58] W2122908584 via openalex

[59] W2132577372 via openalex

[60] W2157568141 via openalex

[61] W166362025 via openalex

[62] W2322085293 via openalex

[63] W2550657053 via openalex

[64] W2891820668 via openalex

[65] W2946091052 via openalex

[66] W2980976550 via openalex

[67] W3028984870 via openalex

[68] W3112220592 via openalex