WITHDRAWN: Integrative Approach Unveils the Therapeutic Potential of Bu-shen-huo-xue recipe in Endometriosis: Insights from Comprehensive Machine Learning, Network Pharmacology, and Single-Cell Analysis
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This study explored the therapeutic potential of the Bu-shen-huo-xue recipe in endometriosis using machine learning, network pharmacology, and single-cell analysis.
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References (46)
- ACOG Committee Opinion No. 760 Summary: Dysmenorrhea and Endometriosis in the Adolescent via openalex
- Chemokine CCL2 enhances survival and invasiveness of endometrial stromal cells in an autocrine manner by activating Akt and MAPK/Erk1/2 signal pathway via openalex
- Chinese Herbal Medicine, Alternative or Complementary, for Endometriosis-Associated Pain: A Meta-Analysis via openalex
- Diagnosis and management of endometriosis via openalex
- Differential Levels of Regulatory T Cells and T-Helper-17 Cells in Women With Early and Advanced Endometriosis via openalex
- Effects of Ulipristal Acetate on Reactive Oxygen Species and Proinflammatory Cytokine Release by Epithelial and Stromal Cells from Human Endometrium and Endometriosis via openalex
- Endometriosis via openalex
- Endometriosis-Associated Ovarian Carcinomas: How PI3K/AKT/mTOR Pathway Affects Their Pathogenesis via openalex
- Epigenetic Dysregulation in Endometriosis: Implications for Pathophysiology and Therapeutics via openalex
- Estrogen receptor β upregulates CCL2 via NF-κB signaling in endometriotic stromal cells and recruits macrophages to promote the pathogenesis of endometriosis via openalex
- Genomic evidence of Y chromosome microchimerism in the endometrium during endometriosis and in cases of infertility via openalex
- Human endometriosis is associated with plasma cells and overexpression of B lymphocyte stimulator via openalex
- Increased Mitogen-Activated Protein Kinase Kinase/Extracellularly Regulated Kinase Activity in Human Endometrial Stromal Fibroblasts of Women with Endometriosis Reduces 3′,5′-Cyclic Adenosine 5′-Monophosphate Inhibition of Cyclin D1 via openalex
- Knockdown of long noncoding RNA CCDC144NL-AS1 attenuates migration and invasion phenotypes in endometrial stromal cells from endometriosis† via openalex
- Macrophages originated IL-33/ST2 inhibits ferroptosis in endometriosis via the ATF3/SLC7A11 axis via openalex
- Pathogenesis Based Diagnosis and Treatment of Endometriosis via openalex
- Pharmaceuticals targeting signaling pathways of endometriosis as potential new medical treatment: A review via openalex
- Rethinking mechanisms, diagnosis and management of endometriosis via openalex
- The expression of estrogen receptors as well as GREB1, c-MYC, and cyclin D1, estrogen-regulated genes implicated in proliferation, is increased in peritoneal endometriosis via openalex
- Transcriptional profiling of endometriosis tissues identifies genes related to organogenesis defects via openalex
- Treatment of endometriosis with dienogest in combination with traditional Chinese medicine: A systematic review and meta-analysis via openalex
- Vascular endothelial growth factor is produced by peritoneal fluid macrophages in endometriosis and is regulated by ovarian steroids. via openalex
- W3135648735 via openalex
- W3202177173 via openalex
- W2008209953 via openalex
- W2006617902 via openalex
- W4307432965 via openalex
- W4309508903 via openalex
- W1985969236 via openalex
- W4377205868 via openalex
- W1982509613 via openalex
- W1977267072 via openalex
- W1966327575 via openalex
- W4393097870 via openalex
- W6846406725 via openalex
- W6852794353 via openalex
- W2159707944 via openalex
- W6862421946 via openalex
- W2169353806 via openalex
- W2489788488 via openalex
- W2747877289 via openalex
- W2800733044 via openalex
- W2092324058 via openalex
- W3016136518 via openalex
- W3132661792 via openalex
- W3134885188 via openalex
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