Environment and Endometriosis: a toxic relationship.

Ilaria Soave, I Soave, D Caserta, Donatella Caserta, J-M Wenger, Jean‐Marie Wenger, S Dessole, Salvatore Dessole, A Perino, Roberto Marci, R Marci
European review for medical and pharmacological sciences · 2015 · vol. 19(11) , pp. 1964–72 · PMID:26125255 · W773586911
article OA: green CC0 ⤵ 21 in-corpus citations
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⚙ AI-generated summary by claude@2026-06, 2026-06-08 ⓘ

This review examines the potential role of environmental dioxins and dioxin-like compounds, which bioaccumulate and persist, in the development of endometriosis.

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Abstract

Endometriosis is a common, benign, estrogen-dependent gynecological disease that represents one of the main causes of hospitalization in industrialized countries. It is well established that a large amount of natural and man-made chemicals are present in the environment and both humans and animals are exposed to them. Dioxin and dioxin-like compounds have long biological half-life, can accumulate within the organism and could negatively affect several physiological processes. The purpose of this review is to provide an overview of the possible relationship between these chemicals and the pathogenesis of endometriosis.

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Condition tags

mesh:D004715endometriosis

MeSH descriptors

Dioxins Endometriosis Environmental Exposure Polychlorinated Biphenyls Animals Dioxins Endometriosis Endometriosis Environmental Exposure Female Humans Polychlorinated Biphenyls

Citation neighborhood

Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

References (15)

Cited by (21)

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europepmc
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License: CC0 · commercial use OK