Additional file 6 of The IL-33-ST2 axis plays a vital role in endometriosis via promoting epithelial–mesenchymal transition by phosphorylating β-catenin

Ruan, Jingyao, Tian, Qi, Li, Siting, Zhou, Xiaoyu, Sun, Qianzhi, Wang, Yuning, Xiao, Yinping, Li, Mingqing, Chang, Kaikai, Yi, Xiaofang
2024 · doi:10.6084/m9.figshare.26731708 · W6977065716
other OA: green CC0
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⚙ AI-generated summary by claude@2026-06, 2026-06-07 ⓘ

This study investigated the IL-33-ST2 axis in endometriosis, finding it promotes epithelial-mesenchymal transition by phosphorylating β-catenin and correlating ST2 expression with vimentin.

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Abstract

Additional file 6: Supplementary Figure 2. Immunohistochemical staining of ST2, Vimentin, and E-cadherin. A Percentage of collagen area to total area in eutopic endometrium and ectopic lesion from controls and EMs patients as determined by Masson staining (Scale bars, 100 μm). B-D Relative mean optical densities of ST2 (IL-33 receptor), vimentin, and E-cadherin as determined by immunohistochemical (IHC) staining (Scale bars, 20 μm). E Simple linear regression of ST2 and vimentin expressions (Y = 0.9617 * X + 0.09133, R2 = 0.8094, P < 0.0001). F Simple linear regression of percentage of collagen deposition and CCN4 expressions in human samples (Y = 0.7490 *X - 53.62, R2 = 0.8088, P < 0.0001). G Simple linear regression of percentage of collagen deposition and CCN4 expressions in mouse sample (Y = 125.6 * X - 21.80, R2 = 0.8704, P < 0.0001).

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endometriosis

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Papers in the corpus that this work cites (lower rings, blue) and that cite this one (upper rings, green). Dot size scales with the paper's in-corpus citation count — bigger dot = more influential within the endo/adeno field. Click a dot to open that paper. [ expand to 2 hops ] — adds papers reached through this work's immediate citers/citees. Heavier; up to 60 extra dots.

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