Maobi Zhu

ORCID: 0000-0002-1700-7917 · 4 papers in corpus
lab-animal 2019
Lipoxin A4 Suppresses IL-1β-Induced Cyclooxygenase-2 Expression Through Inhibition of p38 MAPK Activation in Endometriosis
Reproductive sciences (Thousand Oaks, Calif.) ·doi:10.1177/1933719119828115

Endometriosis is an inflammation-dependent gynecologic disorder. Increased cyclooxygenase-2 (COX-2) expression plays an important role in the development and progression of endometriosis. Lipoxin A4 (LXA4) is an endogenous anti-inflammation…

lab-animal 2017
Dioxin-like rather than non-dioxin-like PCBs promote the development of endometriosis through stimulation of endocrine-inflammation interactions
Archives of toxicology ·doi:10.1007/s00204-016-1854-0

Polychlorinated biphenyls (PCBs) contain 209 congeners with various structure-activities. Exposure to PCBs was related to disorders of female reproduction. Endometriosis (EM) is an estrogen- and inflammation-dependent disease with high prev…

other 2014
Lipoxin A4 regulates expression of the estrogen receptor and inhibits 17β-estradiol induced p38 mitogen-activated protein kinase phosphorylation in human endometriotic stromal cells
Fertility and sterility ·doi:10.1016/j.fertnstert.2014.03.029

OBJECTIVE: To study the role of lipoxin A4 (LXA4) in endometriosis. DESIGN: Molecular analysis in human samples and primary human endometriotic stromal cells (ESCs). SETTING: University hospital. PATIENT(S): Forty-nine premenopausal wome…

lab-animal 2014
Lipoxin A4 suppresses the development of endometriosis in an ALX receptor-dependent manner via the p38 MAPK pathway
British journal of pharmacology ·doi:10.1111/bph.12816

BACKGROUND AND PURPOSE: Lipoxins can function as endogenous 'breaking signals' in inflammation and play important roles in the progression of endometriosis. In this study, we further investigated the molecular mechanism by which lipoxin A4 …