Ting Geng

No ORCID on file · 10 papers in corpus · active 2020-2026

Study types

  • article 5
  • preprint 3
  • other 2

Condition tags

  • endometriosis 9
  • mesh:D004715 3
  • infertility 3
preprint 2026
Endometrial cells with high ALDH activity contribute to uterine development and regeneration
·doi:10.21203/rs.3.rs-6831916/v3
other 2026
Endometrial epithelial cells with high ALDH activity control uterine development and regeneration
Research square ·doi:10.21203/rs.3.rs-6831916/v2

Adult stem cells are thought to drive the regenerative potential of the endometrium and contribute to the pathogenesis of endometriosis, however, their identity and defining features remain to be characterized. Here, we used in vivo and in …

article 2026
Endometrial epithelial cells with high ALDH activity control uterine development and regeneration
·doi:10.7554/elife.110975.1

Adult stem cells are thought to drive the regenerative potential of the endometrium and contribute to the pathogenesis of endometriosis, however, their identity and defining features remain to be characterized. Here, we used in vivo and in …

article 2026
Endometrial epithelial cells with high ALDH activity control uterine development and regeneration
·doi:10.7554/elife.110975

Adult stem cells are thought to drive the regenerative potential of the endometrium and contribute to the pathogenesis of endometriosis, however, their identity and defining features remain to be characterized. Here, we used in vivo and in …

preprint 2025
ALDH1A1+ cells are endometrial progenitors contributing to regeneration and endometriosis
·doi:10.21203/rs.3.rs-6831916/v1
article 2024
Impaired bone morphogenetic protein (BMP) signaling pathways disrupt decidualization in endometriosis
Communications biology ·doi:10.1038/s42003-024-05898-z

Endometriosis is linked to increased infertility and pregnancy complications due to defective endometrial decidualization. We hypothesized that identification of altered signaling pathways during decidualization could identify the underlyin…

other 2023
Impaired bone morphogenetic protein (BMP) signaling pathways disrupt decidualization in endometriosis
Research square ·doi:10.21203/rs.3.rs-3471243/v1

Endometriosis is linked to increased infertility and pregnancy complications due to defective endometrial decidualization. We hypothesized that identification of altered signaling pathways during decidualization could identify the underlyin…

preprint 2023
Impaired bone morphogenetic protein signaling pathways disrupt decidualization in endometriosis
bioRxiv : the preprint server for biology ·doi:10.1101/2023.09.21.558268

ABSTRACT It is hypothesized that impaired endometrial decidualization contributes to decreased fertility in individuals with endometriosis. To identify the molecular defects that underpin defective decidualization in endometriosis, we subje…

article 2022
Downregulation of LHCGR Attenuates COX-2 Expression and Induces Luteinized Unruptured Follicle Syndrome in Endometriosis
Frontiers in endocrinology ·doi:10.3389/fendo.2022.853563

An association between endometriosis and luteinized unruptured follicle syndrome (LUFs) has long been identified. Although inactivating mutation of luteinizing hormone/choriogonadotropin receptor (LHGCR) results in LUFs, whether LHCGR contr…

article 2020
Decreased mixed lineage leukemia 1 is involved in endometriosis-related infertility
Journal of molecular endocrinology ·doi:10.1530/jme-20-0193

The aberrant histone methylation patterns contribute to the pathogenesis of endometriosis (EM). Mixed lineage leukemia 1 (MLL1), a histone methyltransferase, is crucial for gene expression by catalyzing the trimethylation of histone 3 lysin…