Retha R. Newbold

No ORCID on file · 5 papers in corpus
lab-animal 2012
Reproductive toxicology (Elmsford, N.Y.) ·doi:10.1016/j.reprotox.2012.08.005

ERΔ3 transgenic mice expressing a dominant negative estrogen receptor α (ERα) variant lacking the second zinc finger in the DNA binding domain were developed to examine its potential to inhibit estrogen action in vivo. To investigate if ERΔ…

lab-animal 2009
Environmental health perspectives ·doi:10.1289/ehp.0800045

BACKGROUND: Exposure to endocrine-disrupting chemicals during critical developmental periods causes adverse consequences later in life; an example is prenatal exposure to the pharmaceutical diethylstilbestrol (DES). Bisphenol A (BPA), an en…

lab-animal 2007
Reproductive toxicology (Elmsford, N.Y.) ·doi:10.1016/j.reprotox.2007.07.006

The developing fetus is uniquely sensitive to perturbation by chemicals with hormone-like activity. The adverse effects of prenatal diethylstilbestrol (DES) exposure are a classic example. Since concern has been mounting regarding the human…

lab-animal 2007
Human Reproduction ·doi:10.1093/humrep/dem185

BACKGROUND: Due to dietary exposure of women to genistein, a soy-derived phytoestrogen, and the estrogen responsiveness of uterine leiomyomas 'fibroids', we evaluated the effects of genistein (0.001-50 microg/ml) on human uterine leiomyoma …

lab-animal 1994
Fertility and sterility ·doi:10.1016/s0015-0282(16)56712-0

OBJECTIVE: To determine the impact of intraperitoneal inflammation on reproduction in the mouse. DESIGN: The effect of an elicited sterile intraperitoneal inflammatory exudate and the passive intraperitoneal transfer of activated syngeneic…