Lídice Mendez-Pérez

No ORCID on file · 3 papers in corpus
lab-animal 2025
Bidirectional Interaction Between PGE2-Preconditioned Mesenchymal Stem Cells and Myofibroblasts Mediates Anti-Fibrotic Effects: A Proteomic Investigation into Equine Endometrial Fibrosis Reversal
Proteomes ·doi:10.3390/proteomes13030041

BACKGROUND: Endometrosis is a prevalent fibrotic condition in mares that impairs reproductive efficiency by inducing transdifferentiation of endometrial stromal cells into myofibroblasts, leading to excessive ECM deposition. METHODS: To el…

lab-animal 2025
PGE2-Primed Mesenchymal Stem Cells Modulate Myofibroblast Function via Bidirectional Communication: A Proteomic Investigation into Equine Endometrial Fibrosis Reversal
·doi:10.20944/preprints202507.0372.v1

Endometrosis is a prevalent fibrotic condition in mares that impairs reproductive efficiency by inducing aberrant transformation of endometrial stromal cells into myofibroblasts, leading to excessive extracellular matrix deposition. To eluc…

lab-animal 2024
Extracellular vesicles secreted by equine adipose mesenchymal stem cells preconditioned with transforming growth factor β-1 are enriched in anti-fibrotic miRNAs and inhibit the expression of fibrotic genes in an in vitro system of endometrial stromal cells fibrosis
The veterinary quarterly ·doi:10.1080/01652176.2024.2384906

Mare endometrosis is a major reproductive problem associated with low fertility and is characterized by persistent inflammation, TGFβ-1 signaling, and consequently, extracellular matrix deposition, which compromises endometrial glands. Mese…